Towards a high-precision atomic gyroscope

Thesis (S.M.)--Massachusetts Institute of Technology, Dept. of Physics, 2013.Cataloged from PDF version of thesis.Includes bibliographical references (pages 69-72).In this thesis, I report on the design and construction of the Rubidium Atomic Gyroscope Experiment (RAGE) at Draper Lab.by Mackenzie A. Van Camp.S.M

Optomechanical Cavity Cooling of an Atomic Ensemble

We demonstrate cavity sideband cooling of a single collective motional mode of an atomic ensemble down to a mean phonon occupation number of 2.0(-0.3/+0.9). Both this minimum occupation number and the observed cooling rate are in good agreement with an optomechanical model. The cooling rate constant is proportional to the total photon scattering rate by the ensemble, demonstrating the cooperative character of the light-emission-induced cooling process. We deduce fundamental limits to cavity-cooling either the collective mode or, sympathetically, the single-atom degrees of freedom.Comment: Paper with supplemental material: 4+6 pages, 4 figures. Minor revisions of text. Supplemental material shortened by removal of supplementary figur

Mid-infrared silicon photonics

A mid-infrared silicon nanophotonic integrated circuit platform can have broad impact upon environmental monitoring, personalized healthcare, and public safety applications. Development of various mid-IR components, including optical parametric amplifiers, sources, modulators, and detectors, is reviewed

Velocity-space sensitivity of the time-of-flight neutron spectrometer at JET

The velocity-space sensitivities of fast-ion diagnostics are often described by so-called weight functions. Recently, we formulated weight functions showing the velocity-space sensitivity of the often dominant beam-target part of neutron energy spectra. These weight functions for neutron emission spectrometry (NES) are independent of the particular NES diagnostic. Here we apply these NES weight functions to the time-of-flight spectrometer TOFOR at JET. By taking the instrumental response function of TOFOR into account, we calculate time-of-flight NES weight functions that enable us to directly determine the velocity-space sensitivity of a given part of a measured time-of-flight spectrum from TOFOR

Relationship of edge localized mode burst times with divertor flux loop signal phase in JET

A phase relationship is identified between sequential edge localized modes (ELMs) occurrence times in a set of H-mode tokamak plasmas to the voltage measured in full flux azimuthal loops in the divertor region. We focus on plasmas in the Joint European Torus where a steady H-mode is sustained over several seconds, during which ELMs are observed in the Be II emission at the divertor. The ELMs analysed arise from intrinsic ELMing, in that there is no deliberate intent to control the ELMing process by external means. We use ELM timings derived from the Be II signal to perform direct time domain analysis of the full flux loop VLD2 and VLD3 signals, which provide a high cadence global measurement proportional to the voltage induced by changes in poloidal magnetic flux. Specifically, we examine how the time interval between pairs of successive ELMs is linked to the time-evolving phase of the full flux loop signals. Each ELM produces a clear early pulse in the full flux loop signals, whose peak time is used to condition our analysis. The arrival time of the following ELM, relative to this pulse, is found to fall into one of two categories: (i) prompt ELMs, which are directly paced by the initial response seen in the flux loop signals; and (ii) all other ELMs, which occur after the initial response of the full flux loop signals has decayed in amplitude. The times at which ELMs in category (ii) occur, relative to the first ELM of the pair, are clustered at times when the instantaneous phase of the full flux loop signal is close to its value at the time of the first ELM

Long-external-cavity distributed Bragg reflector laser with subkilohertz intrinsic linewidth

We report on a simple, compact, and robust 780 nm distributed Bragg reflector laser with subkilohertz intrinsic linewidth. An external cavity with optical path length of 3.6 m, implemented with an optical fiber, reduces the laser frequency noise by several orders of magnitude. At frequencies above 100 kHz the frequency noise spectral density is reduced by over 33 dB, resulting in an intrinsic Lorentzian linewidth of 300 Hz. The remaining low-frequency noise is easily removed by stabilization to an external reference cavity. We further characterize the influence of feedback power and current variation on the intrinsic linewidth. The system is suitable for experiments requiring a tunable laser with narrow linewidth and low high-frequency noise, such as coherent optical communication, optical clocks, and cavity QED experiments

A dominant-negative GFI1B mutation in the gray platelet syndrome

Contains fulltext : 127547.pdf (publisher's version ) (Open Access)The gray platelet syndrome is a hereditary, usually autosomal recessive bleeding disorder caused by a deficiency of alpha granules in platelets. We detected a nonsense mutation in the gene encoding the transcription factor GFI1B (growth factor independent 1B) that causes autosomal dominant gray platelet syndrome. Both gray platelets and megakaryocytes had abnormal marker expression. In addition, the megakaryocytes had dysplastic features, and they were abnormally distributed in the bone marrow. The GFI1B mutant protein inhibited nonmutant GFI1B transcriptional activity in a dominant-negative manner. Our studies show that GFI1B, in addition to being causally related to the gray platelet syndrome, is key to megakaryocyte and platelet development

A dominant-negative GFI1B mutation in the gray platelet syndrome

Contains fulltext : 127547.pdf (publisher's version ) (Open Access)The gray platelet syndrome is a hereditary, usually autosomal recessive bleeding disorder caused by a deficiency of alpha granules in platelets. We detected a nonsense mutation in the gene encoding the transcription factor GFI1B (growth factor independent 1B) that causes autosomal dominant gray platelet syndrome. Both gray platelets and megakaryocytes had abnormal marker expression. In addition, the megakaryocytes had dysplastic features, and they were abnormally distributed in the bone marrow. The GFI1B mutant protein inhibited nonmutant GFI1B transcriptional activity in a dominant-negative manner. Our studies show that GFI1B, in addition to being causally related to the gray platelet syndrome, is key to megakaryocyte and platelet development